A new report in the Journal of the American Planning Association concludes that density doesn’t make a city sick; crowding and connectivity do. It’s important to disentangle those concepts.
The virus responsible for Covid-19 can survive for up to 28 days on surfaces such as glass, steel, vinyl, paper and polymer banknotes, Australian researchers said on Monday, reinforcing the importance of effective cleaning and handwashing to curb the spread of the disease.
Sweden’s top health authority says people who have had the novel coronavirus are likely to be immune for at least six months after being infected, whether they’ve developed antibodies or not.
Two new studies, though from different parts of the world, have arrived at the same conclusion: that young children not only transmit SARS-CoV-2 efficiently, but may be major drivers of the pandemic as well.
In the midst of a global crisis, scientists are trying to solve an epistemologically intractable question. Defining whether a drug “works” has never been easy, a task vexed by methodological uncertainty, commercial pressures, statistical errors, or sometimes straight-out bad practices. Facing a new disease, researchers have to rethink what success even means. Is it lower mortality? Less disability upon recovery? Faster recovery? The answers are cryptic because the questions are just educated guesses.
This paper develops and implements a method to monetize the impact of moderate social distancing on deaths from COVID-19. It measures the number of deaths saved with the united states governments value of statistical life and finds that mortality benefits of social distancing is 18 trillion dollars
Variations at two spots in the human genome are associated with an increased risk of respiratory failure in patients with Covid-19, the researchers found. One of these spots includes the gene that determines blood types. Having Type A blood was linked to a 50 percent increase in the likelihood that a patient would need to get oxygen or to go on a ventilator, according to the new study.
Neuroscientist Karl Friston, of University College London, builds mathematical models of human brain function. Lately, he’s been applying his modelling to Covid-19. He says, our approach, which borrows from physics and in particular the work of Richard Feynman, goes under the bonnet. It attempts to capture the mathematical structure of the phenomenon – in this case, the pandemic – and to understand the causes of what is observed.
Shutdown orders prevented about 60 million novel coronavirus infections in the United States and 285 million in China, according to a research study published Monday that examined how stay-at-home orders and other restrictions limited the spread of the contagion.
More information is needed after a French study suggested nicotine might play a role in preventing some illness from the coronavirus. Nicotine is not thought to attack sars-cov-2 directly. It may, however,
play an indirect role that involves a cell-membrane protein called ace2,
to which the virus attaches itself in order to gain access to a cell.
Some researchers suspect that nicotine binds to ace2 as well, and that
this makes it harder for the virus to do so alongside it.